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It's more "background enhancement" than an acute stimulant the effect builds over a cycle

The 250500 mcg subcutaneous range used in current research contexts sits below the 1 mg clinical trial dose, partly because subcutaneous bioavailability differs from the oral administration used in some trial arms

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The satiety effect is additive with GLP-1 agonism because the pathways don't overlap which is why CagriSema outperforms semaglutide monotherapy and tirzepatide on some endpoints