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Flavonoids have emerged as promising therapeutic candidates, offering the potential to target both neuroinflammation and oxidative stress simultaneously ( 2.1 The interplay of neuroinflammation and oxidative stress in epilepsy Neuroinflammation plays a crucial role in epilepsy progression

Subcellular distribution of superoxide dismutases (SOD) in rat liver: Cu,Zn-SOD in mitochondria

Moreover, ECM1 interacts with connective tissue growth factor (CTGF) to inhibit integrin v6-mediated activation of TGF, preventing liver fibrosis and ductular reaction (DR) in alcohol-associated liver disease (ALD) and cholangiopathy [14]

The modifications, however, were not regarded as clinically important

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