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[14] first characterized this cell death by iron dependent lipid peroxidation leading to disrupt membrane properties and shrunken mitochondria

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282C1113C1120

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I., Guven, H., Ozgun, H

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The exposure of HaCaT cells to 0, 0.1, 0.25, 0.5, or 1 mg/mL GSH for 72 h resulted in the concentration-dependent induction of apoptosis (Figure 2b,c)

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Reduced somatostatin-like immunoreactivity in cerebral cortex from cases of Alzheimer disease and Alzheimer senile dementa

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