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Based on previous findings regarding the activities of GSH, we hypothesized that GSH would promote fat graft survival by suppressing ferroptosis

Additionally, we noted the inhibition of cell death with Nec-1, suggesting that OGD has the potential to induce necroptosis, aligning with findings by Qi et al

[1] [2] These medications work by mimicking the natural GLP-1 hormone, which enhances insulin secretion, suppresses glucagon release, slows gastric emptying, and reduces appetite

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