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Screening in ALS and FTD patients reveals 3 novel UBQLN2 mutations outside the PXX domain and a pure FTD phenotype

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Compared with 12% of PTEN loss in CM where deletions and mutations both account for the changes (79), PTEN is deeply deleted in merely 6.0% of MM cases and has hardly any mutations ( PTEN and amplified KIT that possibly implies that loss of PTEN or gain of KIT are redundant for the activation of the PI3K pathway in MM

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