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In addition, simultaneous blockade of both mGlu5 and A2AR in A2AmGlu5-D2R RM increases their efficacy in reversing parkinsonian deficits (29)

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Recent studies indicate that antidiabetic drug therapies can enhance neuronal survival and improve cognitive functions in diabetes by improving BBB integrity, reducing inflammation markers, limiting oxidative stress [8,9,10], and increasing the levels of neurotrophins [11]

Meal intake decreased bilateral insula activation, and GLP-1R antagonist reversed the meal-induced reductions in the bilateral insula activity in response to high-energy food, in addition to leading to increased hunger scores (Jennifer et al., 2015)